The most studied healing peptide. Repairs tendons, ligaments, gut lining and more at the local level.

Systemically active — no need to inject near the injury. The ideal partner to BPC-157 for whole-body recovery.

Tiny tripeptide, massive gut impact. Enters gut epithelial cells directly to shut down NF-kB inflammation.

Modulates over 4,000 human genes. Declines with age. A cornerstone of both healing and longevity protocols.

The weight loss revolution in a syringe. STEP trials showed results rivaling bariatric surgery. SELECT trial proved cardiovascular protection.

SURMOUNT-1 showed 22.5% average body weight loss — the most effective anti-obesity drug ever studied until Retatrutide.

The next frontier. Phase 2 data: 24.2% body weight loss at 48 weeks — the highest ever recorded in a pharmacological trial.

Amylin analogue studied in combination with semaglutide (CagriSema). One of the most closely watched obesity drug trials in development.

HGH's fat-burning fragment without any of the GH side effects. Zero IGF-1 impact — pure targeted lipolysis.

A small molecule NNMT inhibitor. Animal studies suggest fat mass reduction and improved insulin sensitivity without diet changes.

The most popular GHRH analogue. With DAC for weekly dosing or without (MOD-GRF 1-29) for natural pulsatile GH release.

The cleanest GH-releasing peptide. No cortisol, no prolactin elevation. The gold standard for beginners and the go-to for sensitive users.

The granddaddy of GHRH analogues. Decades of clinical use. Stimulates pituitary GH release naturally and preserves feedback mechanisms.

FDA approved and the most clinically proven GHRH analogue. Reduces visceral fat 15–20% and may protect cognitive function in adults 40+.

The only oral GH secretagogue. 24-hour half-life. Perfect for those who prefer not to inject. Significant appetite side effect to manage.

Developed in Russia for stroke and ADHD. Dramatically upregulates BDNF — the brain's primary growth factor. Crosses the BBB in minutes via intranasal route.

N-acetylated form of Semax — improved stability and bioavailability, ideal for SubQ injection. Same powerful BDNF and cognitive effects.

Non-addictive anxiolytic without benzo sedation or dependence. The ideal daytime complement to Semax — anxiety relief without the fog.

N-acetylated Selank for improved SubQ bioavailability. Same non-addictive anxiolytic and nootropic effects with better stability.

The original sleep peptide. Named for inducing deep slow-wave (delta) sleep. Also modulates the hormonal stress response.

The cellular energy currency that declines with age. SubQ NAD+ is replacing expensive IV clinics for mitochondrial restoration and anti-aging.

The only FDA-approved peptide for sexual dysfunction. Works from the brain down — creates desire, not just blood flow. Game-changing for both men and women.

The 'bonding hormone' — roles in trust, attachment, social cognition, and anxiety. Researched for autism, PTSD, eating disorders and more.

The master upstream regulator of the entire reproductive hormonal axis. Its discovery revolutionized understanding of puberty and fertility.

The precursor to PT-141. Non-selective melanocortin agonist — tanning, arousal, appetite suppression in one. Higher risk profile than its derivatives.

The only compound shown in peer-reviewed research to lengthen human telomeres in vitro. 30+ years of Khavinson research. The cornerstone longevity peptide.

Restores thymic function and T-cell maturation. Khavinson's 35-year geriatric study showed significant mortality reduction. The thymus shrinks after adolescence — Thymalin reverses this.

Approved as Zadaxin in 40+ countries for hepatitis B/C and cancer adjunct. One of the most clinically validated immune peptides in existence.

The only human cathelicidin. Destroys MRSA and VRSA through a resistance-proof membrane disruption mechanism. First-line antimicrobial defense.

The body's master antioxidant. Declines with age and illness. SubQ delivery is replacing expensive IV sessions for antioxidant replenishment and detox.

Encoded in mitochondrial DNA. Discovered 2015 at USC. Animal studies showed exercise-equivalent fitness gains in sedentary subjects. Activates AMPK — the master metabolic switch.

Concentrates 1000x in the inner mitochondrial membrane. Protects cardiolipin from oxidative damage. Currently in Phase 2/3 trials for heart failure.

2023 Washington University discovery. Rat studies: ~70% endurance increase in 4 weeks without exercise. The most exciting exercise mimetic in research today.

Activates tissue protection without stimulating red blood cell production. Uniquely regenerates small fiber nerves — highly relevant for diabetic and long COVID neuropathy.

Mimics LH to stimulate testosterone production and preserve testicular function during TRT. One of the few peptides with long-standing FDA approval.

The safer melanocortin. Selectively binds MC1R for melanin production without the arousal/appetite effects of MT-2. EU-approved for the painful genetic disorder EPP.

The most potent anabolic peptide in research. Two structural modifications give it a 20–30 hour half-life vs minutes for native IGF-1 — driving continuous muscle hyperplasia, satellite cell activation, and nutrient partitioning that HGH alone can't match.

The original 'Botox in a bottle.' A synthetic hexapeptide that mimics the N-terminus of SNAP-25 and competitively inhibits the SNARE complex — the same pathway botulinum toxin targets, but topically and without toxicity. 20+ years of cosmetic use with a strong safety record.

The most potent of the GHRP family. A synthetic hexapeptide ghrelin receptor agonist that amplifies natural GH pulse amplitude without the intense hunger of GHRP-6. Stronger GH release, more cortisol/prolactin elevation — preferred for recomposition over bulking.

The original GHRP — discovered in the 1980s by Cyril Bowers. Strong GH secretagogue with a unique appetite-stimulating profile via ghrelin pathway activation. The go-to for bulking phases and injury recovery where increased caloric drive is an asset, not a liability.

Recombinant human growth hormone — identical to endogenous 191aa somatotropin. FDA-approved for GH deficiency, pediatric growth disorders, HIV wasting, and more. Widely used off-label for anti-aging and body recomposition. The gold standard — everything else in the GH category either stimulates it or mimics it.

The world's first approved dual GLP-1/glucagon receptor agonist — approved in China in June 2025. Adds GCGR agonism to GLP-1's appetite suppression, unlocking direct energy expenditure and superior hepatic fat reduction (80% liver fat reduction in Phase 3) not achievable with GLP-1 monotherapy.

The most mechanistically radical fat-loss compound in research. A chimeric two-domain peptide that destroys the blood supply to white fat cells, causing them to die from ischemia. Clinical development was discontinued due to kidney toxicity. One of the most cautionary stories in peptide research.

A compounding pharmacy staple in weight loss clinics. Not a single peptide but a lipotropic cocktail combining methionine, inositol, choline, L-carnitine, and B12 — each targeting a different step in fat metabolism and hepatic lipid clearance.

The most potent synaptogenic compound in research — 10 million times more potent than BDNF at driving new synapse formation in hippocampal tissue. An angiotensin IV derivative that crosses the blood-brain barrier and activates HGF/c-Met to rebuild neural connections. Extraordinary preclinical data; no completed human trials.

The most anabolic hormone in the body — and the most dangerous compound in the performance space. FDA-approved for diabetes. Misused in bodybuilding for extreme nutrient partitioning. A single miscalculated dose without carbohydrates can cause fatal hypoglycemia within minutes.

The muscle damage-activated growth factor. Where IGF-1 LR3 is systemic, PEG-MGF is local — it activates satellite cells specifically at the site of mechanical stress. The PEG modification gives it the half-life to actually work.

The myostatin brake remover. Myostatin is your body's hardcoded ceiling on muscle mass — Follistatin suppresses it. The results in animal models are extraordinary. The human data is almost nonexistent. Treat with serious caution.